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. 2018 Apr 13;9:1461. doi: 10.1038/s41467-018-03687-x

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Crossreactive antibodies inhibit ELR⁺ CXC chemokine signaling in vitro. Residual activity of a human hCXCL1, hCXCL5, and hCXCL8 and b mouse mCXCL1 and mCXCL2 chemokines incubated with varying concentrations of SA129 (red), SA138 (blue), and SA157* (gray) fusions, and commercial neutralizing antibodies (Ab, white). The indicated values are the means of three independent experiments. c Plot displaying pK_i versus the calculated pK_D of SA129 (red), SA138 (blue), and SA157* (gray) fusions. Data are presented as mean (dots) ± s.e.m. (bars)