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. 2018 Feb 28;5(4):486–492. doi: 10.1002/acn3.524

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© 2018 The Authors. Annals of Clinical and Translational Neurology published by Wiley Periodicals, Inc on behalf of American Neurological Association.

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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(A) Study design and participant information. Details regarding study design and participant recruitment for each subject group. OI, orthostatic intolerances; POTS, postural orthostatic tachycardia syndrome; NMS, neurally mediated syncope; anti‐gAChR Abs, antiganglionic acetylcholine receptor antibodies. (B) Anti‐gAChR α3 and β4 antibodies in sera from patients with postural orthostatic tachycardia syndrome (POTS). Anti‐gAChR α3 and β4 antibodies were assessed using luciferase immunoprecipitation system (LIPS) assays. The dotted line indicates the cut‐off. We tested sera from healthy controls (HC), as well as from patients with POTS and other neurologic diseases (OND). Autoantibodies against anti‐gAChR were detected in 29% (10 of 34) samples from patients with POTS.