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. 2018 Apr 1;2018:1483041. doi: 10.1155/2018/1483041

Table 2.

The effect of thymoquinone on antioxidant enzymes in doxorubicin-mediated cardiotoxicity in the mice.

Groups and treatments CAT (nmole of H2O2 consumed/min/mg/protein) SOD (nmol epinephrine protected from oxidation/min/mg protein) GPx (nmol NADPH oxidized/min/mg/protein) GR (nmol NADPH oxidized/min/mg protein) GST (nmol CDNB conjugate/min/mg protein)
Group A (control) 35.93 ± 2.4 12.51 ± 1.56 160.05 ± 3.10 415.89 ± 1.34 159.79 ± 4.56
Group B (DOX) 15.34 ± 2.60 5.29 ± 1.78∗∗∗ 83.94 ± 3.72∗∗ 217.38 ± 2.34∗∗ 80.26 ± 4.21∗∗
Group C (DOX + Q10) 38.23 ± 2.30 8.45 ± 1.58∗∗ 115.34 ± 2.14∗∗ 384.13 ± 2.13 120.46 ± 3.54
Group D (DOX + TQ-20) 34.45 ± 2.11∗∗ 10.34 ± 1.36∗∗∗ 143.67 ± 2.13 420.22 ± 1.65∗∗∗ 159.87 ± 2.45∗∗∗
Group E TQ-20 30.23 ± 1.23∗∗ 13.56 ± 2.10∗∗ 167.45 ± 1.46 428.00 ± 1.34 156.08 ± 2.33∗∗

Each value is the mean of six replicates with standard deviation; p < 0.05, ∗∗p < 0.01, and ∗∗∗p < 0.001 were significant when compared to normal control (Group A) and toxic control (Group-B) by performing Tukey–Kramer test.