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. 2000 Jul;123(3):939–948. doi: 10.1104/pp.123.3.939

Table I.

Metabolization and ability to repress α-amylase induction of the disaccharides used in this study

Compound Chemical Structure Metabolized α-amylase Repression
Suc Glc[1→2]Fru Yes Yes
Turanose Glc[1→3]Fru No Yes
Nigerose Glc[1→3]Glc Yes Yes
3α-Galactobiose Gal[1→3]Gal No No
Cellobiose β-Glc[1→4]Glc Yes Yes
Lactulose β-Gal[1→4]Fru No Yes
Lactitol β-Gal[1→4]Glucitol No No
Lactose β-Gal[1→4]Glc No No
4β-Galactobiose β-Gal[1→4]Gal No No
Leucrose Glc[1→5]Fru Yes Yes
Isomaltose Glc[1→6]Glc Yes Yes
Gentiobiose β-Glc[1→6]Glc Yes Yes
Palatinose Glc[1→6]Fru No Yes
Palatinitol Glc[1→6]Glucitol (50%) No No
Glc[1→6]Mannitol (50%)
Melibiose Gal[1→6]Glc No No

Metabolism is defined as the ability of the disaccharides to induce an increase in the endogenous content of Glc+Fru+Suc equal to or exceeding two times that of control, as well as in bringing about a significant (two times that of control) increase in the dry wt of the isolated embryos. Data on α-amylase repression are based on the ability of the tested disaccharides (80 mm) to repress α-amylase induction by at least 70%.