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. 2018 Mar 4;22(5):2791–2803. doi: 10.1111/jcmm.13572

Figure 2.

Figure 2

Effects of qiliqiangxin (QL) on ROS production and mitochondria membrane potential. (A) QL reduced ROS generation in cardiac microvascular endothelial cells (CMECs) exposed to hypoxia for 12 h. Dihydroethidium (DHE) staining was used to detect ROS level in CMECs (20×, Scale bar 50 μm) (n = 4/group). (B) QL attenuated mitochondria dysfunction in CMECs exposed to hypoxia for 12 h. Mitochondrial membrane potential was detected by JC‐1 staining (10×, Scale bar 100 μm) (n = 4/group). Data were expressed as means ± SD. *P < .05; **P < .01