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. 2018 Mar 27;8:72. doi: 10.1038/s41398-018-0120-z

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a AVPR1a SNPs (as represented by columns) were genotyped and tested for association with DAE (SNP-specific p values are below each column). The SNPs were also considered as haplotypes (indicated on the right Y axis with haplotype frequency increasing from light to dark red), which were grouped into related clusters based on their phylogeny. Three major AVPR1a SNP haplotype clades exist in our DAE sample although in order to compare the effects of the other clusters on DAE, one cluster was split into C2 and C4. b The majority of samples had the clade indicated by C1, so the effects of the other clusters could be compared. The SNP haplotype clusters were not associated with DAE (p = 0.950)