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. 2018 Apr 27;9(5):469. doi: 10.1038/s41419-018-0482-4

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a, b SW1783 and LN229 cells were cotransfected with pCNA3.1-FoxM1 and sh-ADAM17 plasmids, and then determined the capacities of migration, adipogenesis and osteogenesis. Scale bar = 100 μm. The data are shown as the mean ± s.d. of three independent experiments. Student’s t test was used to determine the significance of the differences between the groups (**P < 0.01, ***P < 0.001, Student’s t test). c The effects of FoxM1 overexpression combined with ADAM17 knockdown on mesenchymal transition were investigated by western blot in SW1783 and LN229 cells. d, e The capacities of migration, adipogenesis and osteogenesis were determined after knocking down FoxM1 and overexpressing ADAM17 in U251MG and U87MG cells. Scale bar = 100 μm. Data are shown as the mean ± s.d. of three independent experiments. Student’s t test was used to determine the significance of the differences between the groups (**P < 0.01, ***P < 0.001, Student’s t test). f The effects of FoxM1 knockdown combined with ADAM17 overexpression on mesenchymal phenotype were investigated by western blot in U251MG and U87MG cells