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. 2018 Mar 5;15(5):6400–6408. doi: 10.3892/ol.2018.8177

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Copyright: © Qi et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — GAs inhibit HepG2 cells proliferation. (A) Dose-depended curve of HepG2 cells following treatment with Gas for 24 h. The cells treated with Gas for 24 h exhibited a significantly increased level of inhibition of HepG2 cells compared with the control group (*P<0.05 vs. control group). (B) Images of HepG2 cells treated with GAs at 24 h captured by light microscopy (magnification, ×200). (Ba) control group and (Bb) HepG2 cells treated with GAs. The cells treated with Gas were decreased in size compared with those in the control group. GAs, ginkgolic acids (the P-value of each two groups depicted in Table I).