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. 2018 Mar 19;15(5):7537–7544. doi: 10.3892/ol.2018.8293

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Copyright: © Zhang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Analysis of the activity of JNK1/2/3, p38 and ERK1/2-MAPK/c-Jun signaling pathways in GEC-BARF1 cells. (A-a, B-a and C-a) Western blotting and (A-b, B-b and C-b) quantitative analysis of JNK1/2/3, p38 and ERK1/2 protein phosphorylation, respectively, in GEC cells. (A-c, B-c and C-c) Western blotting and (A-d, B-d and C-d) quantitative analysis of c-Jun expression and phosphorylation in SGC-BARF1 treated with SP600125, SB203580 and U0126 compared with DMSO, respectively. Data were expressed as the mean ± standard deviation from three independent experiments. *P<0.05, **P<0.01 vs. GEC-SG. ^#P<0.05, ^##P<0.01 vs. DMSO control. DMSO, dimethyl sulfoxide.