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. Author manuscript; available in PMC: 2018 Oct 18.
Published in final edited form as: Nature. 2018 Apr 18;556(7702):520–524. doi: 10.1038/s41586-018-0046-x

Extended Data Table 4.

IP accumulation assays of wild-type (WT) and mutant Y1Rs for NPY/NPY analogues

a. IP accumulation assays of complementary mutagenesis between NPY/NPY analogues and WT and mutant Y1Rs
Peptides* WT Q 1203.32H I293ECL3N N299732A N2836.55A D2876.59A
EC50 (nM) (pEC50 ± SEM) X-fold over WT n§ EC50 (nM) (PEC50 ± SEM) X-fold over WT n EC50 (nM) (PEC50 ± SEM) X-fold over WT n EC50 (nM) (PEC50 ± SEM) X-fold over WT n EC50 (nM) (PEC50 ± SEM) X-fold over WT n EC50 (nM) (PEC50 ± SEM) X-fold over WT n
NPY 1.5 (8.83 ± 0.02) 1 51 39 (7.41 ±0.11) 26 3 169 (6.77 ± 0.08) 113 8 91 (7.04 ±0.09) 61 5 1,053 (5.98 ±0.13) 702 6 1,384 (5.86 ±0.09) 461 3
[N30]NPY 289 (6.54 ± 0.06) 1 9 1 / / 3116; 1274 11; 4 2 / / / / / / / / /
[A33]NPY 5,395 (5.27 ± 0.07) 1 3 / / / / / / 5,306 (5.28 ± 0.07) 1 3 / / / / / /
[A35]NPY >10,000 1 3 / / / / / / / / / / / / >4,900 >5 3
[A36]NPY 1,378 (5.86 ± 0.06) 1 6 / / / / / i / / / nd nd 3 / / /
NPY-tyramide 68 (7.17 ±0.11) 1 9 nd nd 3 / i i / / / / i / / / /
b. IP accumulation assays of WT Y1R for NPY/NPY analogues
NPY Ac-NPY [A1]NPY [A2]NPY [A1,A2]NPY NPY(3-36) NPY(13-36)
EC50 (nM) (pEC50 ± SEM) 1.5 (8.83 ± 0.02) 0.5; 3.0 6.5; 2.4 7.3 (8.14 ±0.11) 8.0 (8.10 ±0.06) 83 (7.08 ± 0.09) 477;744
X-fold over NPY 1 1; 2 4; 2 5 5 55 318; 496
n 51 2 2 4 3 3 2
c. NPY-induced IP accumulation assays of WT and mutant Y1Rs
WT F184ECL2A F184ECL2N V187ECL2N L189ECL2N Y192ECL2S V197ECL2A V197ECL2N F199ECL2N F202ECL2N
EC50 (nM) (pEC50 ± SEM) 1.5 (8.83 ± 0.02) 18.7 (7.73 ± 0.08) 23 (7.64 ±0.10) 1.9 (8.72 ± 0.08) 1.9 (8.73 ± 0.16) 3.8 (8.42 ± 0.11) 1.9 (8.71 ± 0.13) 188 (6.73 ± 0.11) 3.4 (8.47 ± 0.11) 1.2 (8.93 ± 0.15)
X-fold over WT 1 13 15 1 1 2.5 1 125 2 1
n 51 7 14 3 3 3 5 7 3 3
*

Peptides were synthesized following the methods described in peptide synthesis section of Methods.

EC50 were determined using GraphPad Prism 5.0. All curves were normalized to the top and bottom values of the Y1R/NPY curve. Nonlinear regression (curve fit) was performed for normalized response in all assays. All data are shown as means from at least three independent experiments (n>2) or results of two individual experiments (n=2) each performed in technical duplicate.

The EC50 shifts were determined by EC50(mutant) / EC50(WT), set Hill slope to 1. For the wild-type receptor x-fold is set to 1. Lower EC50 shift of NPY analogue/mutant compared to NPY/mutant was interpreted as no further loss of function and a direct interaction between both positions.

§

Sample size n, the number of independent experiments performed in technical duplicate.

Data are from reference 27.

nd, not determined up to 10−4 M agonist concentration; /, not tested.