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. 2018 Mar 2;15(5):6291–6296. doi: 10.3892/ol.2018.8162

Figure 2.

Figure 2.

(A) 24-h exposure to PTX and blue light markedly enhanced the apoptotic rate of HL60 cells. (B) PTX and blue light induced the dissipation of mitochondrial membrane potential, detected via JC-1 staining. (C) 24-h exposure to PTX and blue light significantly enhanced intracellular ROS levels in HL60 cells. Data are expressed as the mean ± standard deviation (n=6). **P<0.01 vs. control cells. PTX, paclitaxel; ROS, reactive oxygen species; JC-1, 5,5′, 6,6′-tetrachloro-1,1′,3,3′tetraethylbenzimidazol-ylcarbocyanine iodide; CTRL, control.