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. 2018 Mar 19;17(5):7156–7162. doi: 10.3892/mmr.2018.8774

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Copyright: © Chen et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Luteolin-7-O-glucoside (LUTG) maintained the redox homeostasis in cardiomyocytes. (A) Effect of LUTG on the viability of cardiomyocytes. (B) Effect of LUTG on the viability of the H/R treated cardiomyocytes. (C and D) Flow cytometry analysis for ROS generation. (E) LDH activity analysis. (F) Antioxidant enzyme CAT activity analysis. Control mock (CK); model group (MG); hypoxia/reoxygenation group (H/R); LUTG (10 and 20 µM) pretreatment groups: (H/R+10, H/R+20). Data were presented as mean ± standard deviation, n=4, *P<0.05 and **P<0.01 vs. CK; ^#P<0.05 and ^##P<0.01 vs. H/R.