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. 2018 Mar 13;39(5):2071–2080. doi: 10.3892/or.2018.6310

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — CD40L-M contributes to inhibition of cell proliferation and cell cycle arrest. (A) Cell proliferation was measured using a CCK-8 assay. (B) Cell cycle distribution. (C) The cell cycle was analyzed with PI staining and FACS. (D) p53 and p21 protein was determined using western blot analysis. GAPDH was used as a loading control. Data are expressed as the means ± SD of three independent experiments. *P<0.05 compared to the controls or negative group.

Figure 2. — CD40L-M contributes to inhibition of cell proliferation and cell cycle arrest. (A) Cell proliferation was measured using a CCK-8 assay. (B) Cell cycle distribution. (C) The cell cycle was analyzed with PI staining and FACS. (D) p53 and p21 protein was determined using western blot analysis. GAPDH was used as a loading control. Data are expressed as the means ± SD of three independent experiments. *P<0.05 compared to the controls or negative group.