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. 2018 Mar 2;118(8):1062–1073. doi: 10.1038/s41416-018-0025-x

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-ShareAlike 4.0 International License, which permits any non-commercial use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. If you remix, transform, or build upon this article or a part thereof, you must distribute your contributions under the same license as the original. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-sa/4.0/.

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Fig. 1 — Clustergram of the signals of the 15 genes used to build the Combo score in myeloma cells of 206 previously untreated patients. a The signals of the 15 probe sets in MMCs of 206 patients, ordered by increasing Combo score, are displayed from low (deep blue) to high (deep red) expression. Among the 15 probe sets, the genes associated with a poor outcome are identified by a red frame. b Combo score in normal bone marrow plasma cells (n = 7), in premalignant plasma cells of patients with monoclonal gammopathy of undetermined significance (MGUS, n = 5), in multiple myeloma cells of patients with intramedullary MM (n = 206), and in human myeloma cell lines (n = 40)