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. 2018 May 4;8:7092. doi: 10.1038/s41598-018-25391-y

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Intensity of immunoprecipitated protein for all groups; apolipoprotein E (a), serum amyloid P component (b), clusterin (c), serum albumin (d), complement component C9 (e), gelsolin (f) and myelin proteolipid protein (g); control (C, n = 5), placebo (P, n = 10), IFN-β on day 1 (I1, n = 10), IFN-β on day 5 (I5, n = 10), etomoxir on day 1 (E1, n = 10) and etomoxir on day 5 (E5, n = 10). All data are presented as the mean ± SEM. Results from the unpaired t-tests showed statistically significant differences in the immunoprecipitated protein intensities between P and E1 and E5 (a–d) and between P and I1 and I5 (e–g). Number of asterisks indicates level of statistical significance (*p < 0.05, **p < 0.01 and ***p < 0.001).