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. 2018 May 4;8:7061. doi: 10.1038/s41598-018-25321-y

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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RELM-β has a minimal impact on airway inflammation. Immune cells were recruited into the airways (collected by bronchoalveolar lavage) in both Retnlb sufficient (wild-type) and deficient (KO) mice (A). Cell populations normalized to cell number showed slight variations in the kinetics between WT (solid circles) and KO (open circles) mice (B). Absolute cell numbers suggest no major differences in innate cell populations at early the early time point although neutrophils were present at the late time point in KO airways (C). RELM-β deficient mice had more T cells, while B cell numbers were similar (C). Data are represented as the mean and SD of n = 4–7 mice/group in one representative study of two independent studies. Data at each time point were compared by two-way ANOVA with Dunnett’s and Sidak’s multiple comparisons test to their naïve controls († and Δ), and between genotypes (*) respectively. One, two, and three symbols represent P < 0.05, P < 0.01, and P < 0.001 correspondingly. N – Naïve.