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Effects of complement-opsonized HIV (C-HIV) on dendritic cells (DCs), natural killer (NK) cells, and T cell phenotype. Complement fragments on HIV’s surface modulate responses in DCs and their cross talk with NK cells to inhibit killing and promote the upregulation of factors associated with immune suppression (PD-1, TIM-3, LAG-3) and susceptibility to infection (CD38, CXCR3, CCR4) on T cells.
