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. 2018 May 8;8:7174. doi: 10.1038/s41598-018-25381-0

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Effects of SE-EE on scopolamine-inflicted cholinergic impairment, lipid peroxidation and oxidative stress in C57/BL6N mice: AChE activity – (a) AChE levels in hippocampus, (b) AChE levels in cerebral cortex. Tacrine (10 mg/kg) was used as positive control; Endogenous level of oxidative stress/antioxidant biomarkers – (c,e,g and i) represents the lipid peroxidation (MDA), Superoxide dismutase (SOD), Catalase (CAT) and Glutathione peroxidase (GPx) levels in the hippocampus respectively; (d,f,h and j) represents the MDA, SOD, CAT and GPx levels in the cerebral cortex of the treated animals, respectively. The data are expressed as mean ± SD (n = 5, pooled biological replicates). One-way ANOVA-Tukey’s multiple comparison test was performed. ^#p < 0.05 compared with the control group; *p < 0.05 other treated groups compared with the scopolamine group.