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. 2018 May 9;8:7367. doi: 10.1038/s41598-018-25668-2

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Reduction of RBM3 impairs wound healing. Scratch wounds were created in confluent cultures of 3T3 fibroblasts and wound closure was imaged in real time under homeostatic conditions. (a and b) Representative images of wounds and superimposed traces of fibroblast migration into the wound area in control (a, blue) and RBM3 knockdown (b, red) conditions. (c) Graph of the extend of wound closure as a function of time in the control (con) and RBM3 knockdown (si) experiments shown in panels a and b. (d) Graph summarizing group data on wound closure in terms of area reclaimed, normalized to scratch length, in control and RBM3 knockdown conditions (con, n = 4; si, n = 3; *p < 0.05 1-tailed t-test).