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. 2017 Dec 11;25(5):841–856. doi: 10.1038/s41418-017-0018-y

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© ADMC Associazione Differenziamento e Morte Cellulare 2017

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, and provide a link to the Creative Commons license. You do not have permission under this license to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 6 — a, b HCT116:p53^+/+, HCT116:p53^−/− cells, HCT116:ACER2^+/+, or HCT116:ACER2^+/+ cells were treated with IR-low, IR-high, or sham, and 24 h later, the cells were subjected to LC/MS/MS analyzes for SPH, S1P, or ceramides. The data represent mean values ± SD of three independent experiments. Statistical analyzes were performed as described in Methods section. *represents p < 0.001 and ^# p < 0.01