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. 2017 Dec 15;33(3):541–551. doi: 10.3904/kjim.2016.334

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Copyright © 2018 The Korean Association of Internal Medicine

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted noncommercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 6. — Changes in antioxidative enzyme activities and level of oxidative DNA damage in benzo[a]pyrene (B[a]P)-induced lung cancer mouse model by carboplatin or hyperoxia treatment. The levels of (A) superoxide dismutase (SOD), (B) glutathione (GSH), and (C) catalase activity as the antioxidant enzymes were measured in the total lung homogenate from mice in each groups. (D) 8-Hydroxy-2-deoxyguanosine (8-OHdG), which has been regarded as a potential marker of oxidative DNA damage was also assayed in lung tissue. Values are expressed as mean ± SE. CON, control group; CAR, carboplatin group; H, hyperoxia group. ^ap < 0.05 compared with the B[a]P + CON group.