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. 2018 May 10;8:7449. doi: 10.1038/s41598-018-25929-0

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Effects of XSECC on cerebral vessels and cerebral blood flow in BCCAO rats. (a) Representative T2 relaxometry images of various group rats. The yellow cover on the sham group indicated ROIs of the bilateral hippocampus (R, right; L, left). (b) Typical CBF maps obtained from ASL at the hippocampus level (−3.8 mm relative to bregma). (c) Representative axial MIP maps (upper panel) and coronal data set images (lower panel) of MRA exhibited relatively distinguished cerebral vessels. ACA, MCA, ICA, PCA and BA were identified in the sham group map (ACA, anterior cerebral artery; MCA, middle cerebral artery; ICA, internal carotid artery; PCA, posterior cerebral artery; BA, basilar artery). The red arrow presented the signal-enhanced and tortuous BA, and the yellow arrow presented the weaken signal of ACA. Quantitation of hippocampal T2 values (d), hippocampal CBF (e), and vascular intensity of ACA, MCA, ICA, PCA, BA (f) and vascular tortuosity of BA (g). ^#P < 0.05, ^##P < 0.01 vs. sham group; *P < 0.05, **P < 0.01 vs. model group (one-way ANOVA followed by LSD’s post hoc test, n = 6 per group).