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Satb1 deletion in Th17 cells after the onset of EAE alleviates clinical evidence of disease in EAE mice. Encephalitogenic Th17 cells were prepared from DLN cells derived from SATB1cKOe mice after immunization and stimulated with MOG peptide and IL‐23. CD4 T cells were transferred into naïve WT mice, and Satb1 deleted by tamoxifen injection after the onset of EAE. Clinical status was monitored for 30 days.
