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. 2018 May 11;9(5):562. doi: 10.1038/s41419-018-0631-9

Fig. 5. FOXM1 was overexpressed in the docetaxel-resistant cancer cells, which contributed to the maintenance of the CSC phenotype.

Fig. 5

a The protein levels of FOXM1 were analyzed in CNE2 and CNE2TR cells, DU145 and DU145-DR cells, and SKOV3 and SKOV3R cells by Western blot. b FOXM1 depletion decreased the expression of CSC-associated molecules ALDH1, NANOG, SOX2 and SHH in CNE2TR, and DU145-DR cells. c The ectopic overexpression of FOXM1 in DU145 or CNE2 cells elevated the expression levels of CSC-associated molecules ALDH1, NANOG, SOX2, and SHH