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. 2018 Mar 29;15(6):8433–8441. doi: 10.3892/ol.2018.8374

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — (A) RT-qPCR was conducted to examine the miR-23a levels in gastric cancer cell lines, compared with normal gastric mucosa epithelial GES-1 cells. **P<0.01 vs. GES-1. Following this, AGS cells were transfected with miR-23a inhibitor or NC inhibitor. (B) RT-qPCR was conducted to examine the miR-23a levels. (C) MTT assay, (D) wound healing assay and (E) Transwell assay were conducted to examine cell proliferation, migration and invasion, respectively. **P<0.01 vs. NC inhibitor. The magnifications for the wound healing and Transwell assay images were ×40 and ×200, respectively. RT-qPCR, reverse transcription-quantitative polymerase chain reaction; NC, negative control; miR, microRNA; OD, optical density.