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. 2018 Apr;13(4):664–672. doi: 10.4103/1673-5374.230292

Figure 6.

Figure 6

Effect of PGE on hippocampal histopathology.

(A) Neurons in the hippocampal CA3 area. The box indicates the analyzed area (Nissl staining, ×100). (A, B) Sham group: The neurons were arranged normally, and Nissl bodies in the cytoplasm were abundant (× 400). (C) VD model group: the number of neurons was substantially reduced, and they were sparsely arranged. Nissl bodies in the cytoplasm were decreased (× 400). (D) 50 mg/kg PGE group (× 400). (E) 100 mg/kg PGE group (× 400). (F) Nimodipine group (× 400). In rats given PGE or nimodipine treatment, the number of neurons was increased, with a greater number of Nissl bodies in the cytoplasm. (G) Quantitation of Nissl-positive cells. Data are shown as mean ± SD (n = 10; one-way analysis of variance followed by Student's t-test). *P < 0.05, vs. sham group; #P < 0.05, vs. VD model group; †P < 0.05, vs. 50 mg/kg PGE group. PGE: Panax ginseng extract; VD: vascular dementia.