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. 2018 May 14;8:7500. doi: 10.1038/s41598-018-25775-0

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Effects of LC27, LC67, and PM on TNBS-induced liver injury in mice. (A) Effects on blood ALT (a), AST (b), TNF-α (c), and LPS levels (d). (B) Effects on liver myeloperoxidase (MPO) (a), malondialdehyde (MDA) (b), TNF-α (c), and LPS levels (d). TNBS, except in the normal control group, was intrarectally administered to mice and test agents [saline, LC5, LC27, LC67, LC68 (2 × 10⁹ CFU/mouse), or sulfasalazine (SUL; 50 mg/kg)] were orally administered for 3 days. LPS was determined using LAL assay kit. All values are shown as the mean ± SD (n = 6). ^#p < 0.05 vs. normal control group. *p < 0.05 vs. group treated with TNBS alone.