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. 2018 May 8;9:458. doi: 10.3389/fphar.2018.00458

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Copyright © 2018 Wei, Wang, Duan and Kong.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Noticeably altered metabolites in two cholestatic injury model groups TAA (intoxicated with thioacetamide) and BDL (bile duct ligation), two HLJDD treatment groups THC (on TAA model) and BHD (on BDL model), and HLJDD administrated healthy rats (HLD). NC, normal control group; FA, fatty acid; 2-HB, 2-hydroxybutyrate; GSH, glutathione; ROS, reactive oxygen species; BCAAs, branch-chained amino acids, leucine, isoleucine and valine; ATP, triphosphate adenosine; SAM, S-adenosyl-methionine; Kreb's cycle, tricarboxylic acid cycle; 2-OG, 2-oxoglutarate; NAG, N-acetylglutamate; NAPG, N-phenylacetyl-glycine. ↑↑/↓↓ and ↑/↓ stands for significantly and partly altered (increased or decreased), respectively.