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. 2018 May 9;12:1183–1193. doi: 10.2147/DDDT.S147104

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© 2018 Wang et al. This work is published and licensed by Dove Medical Press Limited

The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed.

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The 10 most contributed residues of alectinib and JH-VIII-157-02 to ALK^WT and ALK^G1202R.

Notes: The most contributed Per-residue interaction spectra of ALK^WT/alectinib (A), ALK^G1202R/alectinib (C), ALK^WT/JH-VIII-157-02 (E), and ALK^G1202R/JH-VIII-157-02 (G); structural analysis of ALK^WT/alectinib (B), ALK^G1202R/alectinib (D), ALK^WT/JH-VIII-157-02 (F), and ALK^G1202R/JH-VIII-157-02 (H).

Abbreviation: ALK, anaplastic lymphoma kinase.