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. 2018 May 15;8:7550. doi: 10.1038/s41598-018-25245-7

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Developmental trajectory of axial diffusivity (AD; L1) changes for all six subregions of the CC ranging from about 26 weeks to 54 weeks postmenstrual age (PMA) in very preterm infants. Axial diffusivity values, also known as λ₁, remained stable over the first few months in the splenium, isthmus, and posterior midbody. Conversely for segments of the anterior half of the CC, AD remained stable or increased initially until around 40 weeks PMA, after which AD decreases.