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. 2018 Apr 24;9(31):22047–22057. doi: 10.18632/oncotarget.25173

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Copyright: © 2018 Chen et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License 3.0 (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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The mRNA levels of miR-33a (A), CTGF (B), Col1A1 (C), and Col3A1 (D) in heart tissues were examined by qPCR among sham rats (Control), MI rats (MI), antagomiR-33a injected MI rats (antagomiR-33a+MI), and mismatch antagomiR-33a injected MI rats (M-antagomiR-33a+MI). (E) The protein levels of CTGF, Col1A1, and Col3A1 in heart tissues were examined by Western blot among sham, MI, antagomiR-33a+MI, and M-antagomiR-33a+MI. Data are expressed as mean ± SD (n = 3). ^*p < 0.05, ^**p < 0.01, ^***p < 0.001, no significance is indicated as “NS”.

The mRNA levels of miR-33a (A), CTGF (B), Col1A1 (C), and Col3A1 (D) in heart tissues were examined by qPCR among sham rats (Control), MI rats (MI), antagomiR-33a injected MI rats (antagomiR-33a+MI), and mismatch antagomiR-33a injected MI rats (M-antagomiR-33a+MI). (E) The protein levels of CTGF, Col1A1, and Col3A1 in heart tissues were examined by Western blot among sham, MI, antagomiR-33a+MI, and M-antagomiR-33a+MI. Data are expressed as mean ± SD (n = 3). ^*p < 0.05, ^**p < 0.01, ^***p < 0.001, no significance is indicated as “NS”.