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. 2018 May 18;19:367. doi: 10.1186/s12864-018-4746-2

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — BRM occupancy in S2 cells analyzed by ChIP-seq with an antibody against the endogenous BRM. a The average distribution of BRM in genes classified according to gene length as indicated in the figure. n = 1357, 7763 and 4798 for long, medium and short genes, respectively. b The pie diagram shows the distribution of BRM peaks in the genome of S2 cells in relation to gene features. c Metagene analysis of RNAPII distribution in the BRM-bound (red, n = 2521) and not bound (blue, n = 11,403) genes. The plot is derived from data from Lam et al. [52]. The antibody used for the RNAPII ChIP-seq experiment was directed against Rpb3. d The average nucleosome density in the BRM-bound and not bound genes. The plot is derived from data from Shi et al. [25]