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. 2018 May 17;15:148. doi: 10.1186/s12974-018-1188-3

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Deletion of TAK1 from myeloid cells resulted in reduced infarct and improved functional outcomes at day 3 post stroke. a TAK1^ΔM mice had reduced neurological deficit scores (NDS). b Decreased turning bias on the corner test. c Increased locomotor activity. d Decreased infarct area at day 3 post stroke. Data is presented as Mean ± SEM, n = 6–8; *p < 0.05, **p < 0.01 (Two-sample t test or Wilcoxon rank-sum test)