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. 2018 Mar 12;29(5):1475–1489. doi: 10.1681/ASN.2017080932

Figure 2.

Figure 2.

Gpr97 deficiency ameliorated renal IRI in mice. (A) Serum creatinine (SCr) concentration in different groups of mice. (B) BUN levels in different groups of mice. (C) Representative micrographs showing the morphology by hematoxylin and eosin (H&E) staining of the kidney from different groups of mice. (D) Quantitative assessment of tubular damage. (E) In situ terminal deoxynucleotidyl transferase–mediated UTP nick-end labeling (TUNEL) assays were performed to assess renal cell death. Nuclei were revealed using 4’,6-diamidino-2-phenylindole (DAPI) staining. (F) Quantitative assessment of the number of cell death (numbers per high-power field [HPF]). *P<0.05 versus sham-operated wild-type mice (n=8); #P<0.05 versus ischemic wild-type mice at the same experimental conditions.