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. 2018 Mar 12;29(5):1475–1489. doi: 10.1681/ASN.2017080932

Figure 3.

Figure 3.

Gpr97 deficiency reduced inflammatory responses in the kidney after renal IRI in mice. (A) The mRNA levels of proinflammatory mediators including IL-1β, IL-6, TNF-α, and monocyte chemoattractant protein-1 (MCP-1) in the kidney from different groups of mice. (B) Representative sections of kidney stained for macrophages from different groups of mice. (C) Data analysis of macrophage infiltrates in the kidney (numbers per high-power field [HPF]) from different groups of mice. (D) Representative sections of kidney stained for neutrophils from different groups of mice. (E) Data analysis of neutrophil infiltrates in the kidney (numbers per HPF) from different groups of mice. *P<0.05 versus sham-operated wild-type mice; #P<0.05 versus ischemic wild-type mice at the same experimental conditions (n=8).