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. 2018 May 29;8:8346. doi: 10.1038/s41598-018-26421-5

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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The regulation of MALAT1 on MyD88/IRAK1/TRAF6 axis. The HAPI cells were transfected with si-MALAT1, pcDNA-MALAT1, si-NC and pcDNA, respectively. (A,D) The levels of H3 and H4 histone acetylation at the promoter of MyD88 were assessed and (B,C,E,F) the protein expressions of MyD88, IRAK1 and TRAF6 were measured. *P < 0.01 vs. si-NC or pcDNA.