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. 2017 Nov 6;9(3):306–316. doi: 10.1007/s12975-017-0577-8

Fig. 6.

Fig. 6

Schematic role of PGZ administered in the early phase after cerebral ischemia. Activation of PPARγ by PGZ inhibits pro-inflammatory responses and promotes neurogenesis in the peri-infarct region. Some NeuN (green)-, GFAP (green)-, or CD31 (red)-positive cells were mature neurons, extended axons, glias, and blood vessels