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. 2018 May 7;596(11):2037–2054. doi: 10.1113/JP275548

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© 2018 The Authors. The Journal of Physiology published by John Wiley & Sons Ltd on behalf of The Physiological Society

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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A, CSps differentiated into cardiac muscle cells directly after transplantation in vivo; B, CSps promoted the formation of cardiac muscle cells through the mechanism of the paracrine effect; C and D, transplantation of CSps in vivo promoted angiogenesis in infarcted myocardial tissue via a paracrine action; C, DiR‐labelled CSps did not overlap with α‐SMA‐labelled vessel; D, BrdU and α‐SMA staining showing the vessels were newly formed. Areas of blood vessels indicated by a star were enlarged as indicated by the arrows. E, contents of growth factors in fluid of the pericardial cavity. ^* P < 0.05 versus Control group.