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. 2018 Apr 20;39(6):2563–2574. doi: 10.3892/or.2018.6385

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 6. — Suppression of ACVR1 plays important roles in miR-384-silenced mediated aggressive phenotype of breast cancer cells. (A) ACVR1 overexpression in MDA-MB-231/miR-384 cells. (B) Cell growth analyzed by MTT assays. (C-J) Colony formation assay, soft agar assay, Transwell migration assay and wound healing assay. Representative images (C, E, G and I) and quantification (D, F, H and J). *P<0.05, **P<0.01. Vector, MDA-MB-231 cells transfected the vector control; miR-384, MDA-MB-231 cells transfected with miR-384-overexpression plasmid; miR-384+ACVR1, cell overexpressing miR-384 and ACVR1.