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. 2018 Mar 28;17(6):7497–7504. doi: 10.3892/mmr.2018.8811

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Copyright: © Qi et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — PL-PTX promotes ovarian cancer cell apoptosis via the caspase-dependent signal pathway. (A) PL-PTX promoted CAOV-3 ovarian cancer cell apoptosis compared with control group. (B) PL-PTX promoted caspase-3 and caspase-9 activity within CAOV-3 cells compared with control group. (C) PL-PTX treatment increased caspase-3 and caspase-9 protein expression levels within CAOV-3 cells compared with control group. (D) Inhibition of caspase-3 activity via a pan-caspase inhibitor, partially decreased PTX- and PL-PTX-induced apoptosis of CAOV-3 cells compared with in the control group. **P<0.01. PL-PTX, pegylated liposomal-paclitaxel.