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. 2018 Mar 28;17(6):7645–7651. doi: 10.3892/mmr.2018.8806

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — TIP5 promotes the migration of HCC cells by activating β-catenin/transcription factor signaling. (A) TIP5 knockdown inhibited the expression of proteins associated with the epithelial-mesenchymal transition. (B) DN β-catenin reversed the morphological changes induced by TIP5. Magnification, ×10. (C) DN β-catenin rescued the migration of HCC cells. Data are presented as the mean ± standard deviation. Magnification, ×10. **P<0.01 vs. pcDNA3.1, ^##P<0.01 vs. myc-TIP5. HCC, hepatocellular carcinoma; TIP5, transcription termination factor-1 interacting protein 5; DN, dominant negative; sh, small hairpin; con, control.