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. 2018 May 22;9(39):25557–25571. doi: 10.18632/oncotarget.25374

Table 5. Significant hyper-methylated genes based on 10% methylation difference between Cancer and Control (-1000 ≤ distance to transcription start site [TSS] ≤ +1000 base-pair DNA).

Gene Regions DMR location Distance To TSS Methylation Difference Gene Role
Start End % p q
GNAS Promoter 57,464,802 57,465,121 624 28.79 3.48E-04 8.05E-04 GNAS mutation associated with colorectal tumorigenesis [30]
Promoter 57,425,903 57,426,055 0 26.79 1.59E-06 9.22E-06
DNMT3A Intron 25,551,093 25,551,226 365 23.78 8.39E-07 5.36E-06 Methylation pathway
MSH6 Intron 48,011,362 48,011,896 270 20.76 2.71E-03 4.28E-03 Lynch Syndrome, MMR
CAMK1 CDS 9,799,262 9,799,361 7,636 15.32 1.06E-04 3.03E-04 Calcium/calmodulin-dependent protein kinase type 1
MSH2 Intron 47,660,208 47,660,258 30,004 14.75 1.08E-04 3.07E-04 Lynch Syndrome, MMR
GNAS Intron 57,416,524 57,416,680 0 14.50 9.23E-04 1.79E-03 Associated with colorectal tumorigenesis
HK3 CDS 176,308,803 176,309,092 17,242 14.45 3.26E-05 1.16E-04 Glucose metabolism pathways
GNAS Promoter 57,426,743 57,427,047 -786 13.72 6.80E-03 9.05E-03 GNAS mutation associated with Cancer
FCN1 CDS 137,804,570 137,805,004 4,803 11.72 7.28E-05 2.23E-04 Associated with Diabetes
HK3 CDS 176,314,364 176,314,772 11,562 11.32 1.07E-02 1.31E-02 Glucose metabolism pathways
NOS3 CDS 150,710,282 150,710,719 10,868 10.69 2.45E-02 2.66E-02 Methylation, oxidative stress

Note: CDS: coding DNA sequence.