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. 2018 Jun 8;16:26. doi: 10.1186/s12964-018-0241-2

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — KL negatively correlated with HIF1α in CRC patients. To support the correlation obtained from in vitro assays, we first examined the mRNA of KL and HIF1α, and observed no significant correlation between KL and HIF1α (a). Then, we performed IHC staining in CRC patients, and observed a negative correlation between KL and HIF1α in protein level (b). Moreover, the correlation was statistically significant, indicating that KL was associated with HIF1α in protein level (c)