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. 2018 Apr 25;12:95. doi: 10.3389/fncel.2018.00095

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Copyright © 2018 Gao, Zhang, Wang, Fan, Yu, Ye, Wang, Gao, Wang, Luo and Tao.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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IL-33/ST2L signaling inverted TBI-induced Cleaved-Caspase-3 (CC-3) increase. (A) IL-33 pretreatment significantly down-regulated CC-3 expression in cortex after TBI (F_(4,25) = 38.17; ^##P < 0.01 vs. Sham group; **P < 0.01 vs. PBS group, *P < 0.05 vs. PBS group). (B) Anti-ST2L pretreatment obviously up-regulated CC-3 expression in ipsilateralcortex after TBI (F_(2,15) = 40.57; ^#P < 0.05 vs. Sham group; *P < 0.05 vs. PBS group). (C,D) Optical densities of the protein bands were quantitatively analyzed, and normalized with loading control β-actin. The data were expressed as means ± SEM (n = 6/group). Experiments are representative of three independent experiments.