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Aß seals leaky vessels. Traumatic brain injury and cerebrovascular insults stimulate Aß production and draw Aß to the vasculature. Aß binds to red blood cells (RBCs), blood proteins, and to iron (Fe) and copper (Cu) ions. These interactions cause Aß to aggregate at the site of hemorrhage or breaches of the BBB. Aß anchors into cell membranes and increases adherence between RBCs and vascular endothelial cells, helping to seal leaks in the vasculature.
