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. 2018 Jun 13;19:454. doi: 10.1186/s12864-018-4830-7

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 8 — Putative model depicting BTH-mediated Foc 4 resistance via Ca²⁺, hormones and ROS signaling, according to the major DEGs that were modulated by BTH. ET, ethylene; RLKs, receptor-like kinases; ROS, reactive oxygen species; CDPKs, calcium-dependent protein kinases; MAPKs, mitogen-activated protein kinases; TFs, transcription factors; ARFs, auxin response factors; PR, pathogenesis related; SAR, systemic acquired resistance