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. 2018 May 22;175(13):2689–2708. doi: 10.1111/bph.14333

Figure 5.

Figure 5

PZC, PZD and PZE suppressed the up‐regulation of TGFβ/Smad signalling in the TGFβ1‐ or AngII‐stimulated HK‐2 cells and podocytes. (A) Protein expression and (B) quantitative analysis of TGFβ/Smad signalling in the HK‐2 cells induced by TGFβ1. (C) Protein expression and (D) quantitative analysis of TGFβ/Smad signalling in the HK‐2 cells induced by AngII. (E) HK‐2 cells stimulated by TGF‐β1 were stained with primary antibody against p‐Smad3. The in situ expression of p‐Smad3 was analysed by Laser scanning confocal microscopes. Representative immunofluorescent staining images of TGF‐β1‐induced p‐Smad3 expression in the different groups. # P < 0.05 compared with control group (n = 6); * P < 0.05 compared with TGFβ1‐ or AngII‐stimulated group (n = 6).