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. 2018 May 1;32(9-10):670–681. doi: 10.1101/gad.311514.118

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© 2018 Newman et al.; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genesdev.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.

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Figure 2. — rnp-2 mutations disrupt eri-6/7 trans-splicing and cause an Eri phenotype. (A) qRT–PCR for trans-splicing of the enhanced RNAi pathway gene eri-6//7 in wild type compared with two different rnp-2 mutants. (B) Eri phenotypes of splicing mutants; eri-6(mg379) was used as a positive control for a strong Eri phenotype.