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. 2018 Jun 18;18:661. doi: 10.1186/s12885-018-4458-7

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — GOLPH3 contributed to the progression of HCC in vivo; a Photographs of tumors dissected from mice (n = 7) injected with LV-GOLPH3-RNAi or LV-Ctrl HCC cells. b Average tumors weight of mice from each group was shown. c Volume growth curves for the tumors formed by the HCC cells transfected with LV-GOLPH3-RNAi or LV-Ctrl. d HE staining and expression of Ki67 and CD31 in tumors tissue formed by the HCC cells transfected with LV-Ctrl or LV-GOLPH3-RNAi. The magnification of images was 400×. All experiments were performed in triplicate with at least three independent experiments. Data were presented as the mean ± SD. P-values were calculated using Student’s t-test, * P < 0.05, ** P < 0.01, *** P < 0.001, significant difference compared with the LV-Ctrl group