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Actin polymerization contributes to enhanced endothelin-1 (ET-1)-induced vasoconstrictor reactivity after chronic hypoxia (CH). Vasoconstrictor responses to ET-1 in Ca2+-permeabilized, endothelium-disrupted pulmonary arteries from control (A) and CH rats (B) in the presence and absence of cytochalasin B (CytB; 10 μM) are shown. Vehicle data are from Fig. 3A. n = 5–6/group. *P < 0.05 vs. CH vehicle.
